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Vitamin E

Fat-soluble vitamins
Antioxidant

Your intake

Today (logged)
0 mg
0% of 15 mg
Stack potential
0 mg
0% of 15 mg
Target
15 mg
FDA Daily Value
Where you are on the ladder0% of target

What each level of vitamin e does

Approximate dose-response bands. Individual response varies — these are starting points, not prescriptions.

  1. Severely lowYOU ARE HERE
    0 mg4.95 mg

    Well below target. Risk of deficiency symptoms tied to antioxidant.

  2. Insufficient
    4.95 mg15 mg

    Below the recommended daily target. Long-term adequacy not assured.

  3. Adequate
    15 mg22.5 mg

    Daily target met. Standard nutritional support for antioxidant.

  4. Therapeutic
    22.5 mg30 mg

    Common for specific health goals. Check the evidence for your situation before sustaining this level.

  5. High
    30 mg1000 mg

    Approaching the tolerable upper limit. Monitor and consider clinical guidance.

  6. Over upper limit
    1000 mg+

    Above the tolerable upper limit. Risk of adverse effects — back off or consult a clinician.

Overview

Lipid-soluble antioxidant that protects polyunsaturated fatty acids in cell membranes and lipoproteins from peroxidation. Eight natural vitamers (alpha/beta/gamma/delta tocopherol and tocotrienol); alpha-tocopherol is preferentially retained by the hepatic alpha-tocopherol transfer protein.

Functions

  • Chain-breaking antioxidant in lipid membranes
  • Protects LDL particles from oxidative modification
  • Modulates platelet aggregation and smooth muscle proliferation
  • Required for normal neurological function

Mechanism

Donates a hydrogen atom from its chromanol ring to neutralise lipid peroxyl radicals, terminating chain reactions in PUFA-rich membranes. The resulting tocopheroxyl radical is recycled by vitamin C and glutathione. Hepatic alpha-tocopherol transfer protein (alpha-TTP) selects alpha-tocopherol for VLDL incorporation; other vitamers are largely excreted.

Benefits

  • Prevents hemolytic anemia in premature infants and AVED (rare genetic alpha-TTP defect)
  • Possible modest reduction in age-related macular degeneration progression with mixed antioxidants (AREDS)
  • Whole-food vitamin E intake associates with lower coronary risk; high-dose supplements do not replicate this
  • Topical alpha-tocopherol has limited evidence for scar improvement

Deficiency

Frank deficiency is rare in healthy adults — fat malabsorption or genetic alpha-TTP defects are the usual cause. Marginal status is more common but symptomatic deficiency is unusual.

Signs
  • Peripheral neuropathy and ataxia
  • Skeletal myopathy
  • Retinopathy
  • Hemolytic anemia (especially in premature infants)
At-risk groups
  • Cystic fibrosis, cholestatic liver disease, abetalipoproteinemia
  • Premature infants
  • Genetic alpha-TTP defects (AVED)

Excess

High-dose alpha-tocopherol supplementation (≥400 IU/day) is associated with increased all-cause mortality in meta-analyses and modestly raises hemorrhagic stroke risk by inhibiting platelets.

Signs
  • Increased bleeding tendency (especially with anticoagulants)
  • Nausea, fatigue, headache at very high doses
  • Possible increased prostate cancer risk (SELECT trial)

Forms

  • d-alpha-tocopherol
    Natural; the form retained by alpha-TTP; ~2× potency of synthetic
  • dl-alpha-tocopherol
    Synthetic racemic mixture; only the d-isomers are biologically active
  • Mixed tocopherols
    Includes gamma/delta; closer to whole-food spectrum
  • Tocotrienols
    Different side chain; emerging cholesterol-lowering and skin-protective evidence

Food sources

  • Sunflower seeds · 1 oz7 mg
  • Almonds · 1 oz7 mg
  • Sunflower oil · 1 tbsp5 mg
  • Avocado · 1 medium4 mg
  • Hazelnuts · 1 oz4 mg
  • Cooked spinach · 1 cup4 mg

Supplement forms

Choose d-alpha-tocopherol with mixed tocopherols (gamma especially) over synthetic dl-alpha-tocopherol. Doses at or near the 15 mg RDA are sufficient for most adults — high-dose mono-alpha is the wrong default.

Bioavailability

Absorbed with dietary fat; about 50% of dietary intake is absorbed under normal conditions. High-dose alpha-tocopherol displaces gamma-tocopherol from tissues, which may be metabolically undesirable. Synthetic dl-alpha is roughly half as bioavailable as natural d-alpha (the RDA already accounts for this).

Longevity relevance

High-dose supplementation does not extend lifespan and may shorten it. Achieving adequacy through nuts, seeds, and unrefined oils is consistent with the strongest dietary-pattern evidence (Mediterranean, MIND diets).

Relationships

Synergies (works better with)
  • Vitamin C · Recycles oxidised tocopheroxyl radical back to active form
  • Selenium · Glutathione peroxidase (selenoprotein) regenerates reduced glutathione for vitamin E recycling
  • Mixed tocopherols/tocotrienols · Broader membrane coverage than alpha alone
Antagonists (competes with / inhibited by)
  • Warfarin and other anticoagulants · Additive bleeding risk via platelet inhibition and vitamin K interference
  • Statins · May blunt cholesterol-lowering effect in some trials
  • Chemotherapy · High-dose antioxidants may reduce treatment efficacy; discuss with oncologist

References

About Vitamin E

Lipid-soluble antioxidant; protects membranes from oxidative damage.

Role
Antioxidant
Daily target
15 mg (DV)
Upper limit
1000 mg
Also called
vitamin e, alpha-tocopherol, alpha tocopherol, d-alpha-tocopherol, mixed tocopherols, tocotrienols

Forms with lower absorption: dl-alpha-tocopherol, synthetic. Prefer better-absorbed forms when supplementing.

Click here to learn more about Vitamin E
Full explainer on Formulate Health — mechanisms, who's commonly deficient, food sources, evidence for supplementation.
How Vitamin E acts on the body

The mechanisms and systems this nutrient feeds. Click any to drill into what runs on it.